receptor tyrosine kinase meaning in Chinese
受体酪氨酸激酶
Examples
- Pre - clinical studies showed that sorafenib inhibited tumor cell proliferation and angiogenesis by blocking serine / threonine and receptor tyrosine kinases which were inseparable with tumor growth
临床前研究显示，索拉非尼作用的2种激酶会影响肿瘤细胞增生及血管生成，而此2种行为为肿瘤生长时所不可或缺。 - The key mechanisms involved are inhibition of protein - phosphotyrosine phosphatases and activation of nonreceptor protein - tyrosine kinases , in an insulin - receptor tyrosine kinase independent fashion
钒在生物体内发挥作用的主要机制是通过蛋白磷酸酪氨酸磷酸酯酶的抑制和非受体蛋白酪氨酸的激活一种与胰岛素受体完全不同的途径发挥作用的。 - Sorafenib , a novel , oral multi - kinase inhibitor , targets on serine / threonine and receptor tyrosine kinases of the tumor cells and vasculature ; blocks the signal transconduction and inhibits tumor growth
摘要索拉非尼是首个口服多激酶抑制药，靶向作用于肿瘤细胞和肿瘤血管上的丝氨酸和（或）苏氨酸及受体酪氨酸激酶，阻断信号传导，抑制肿瘤生长。 - The neurotrophic and morphogenic activities of gdnf are mediated by its interaction with a multicomponent receptor complex formed by the ret receptor tyrosine kinase and a glycosylphosphatidylinositol ( gpi ) - anchored " accessory " receptor , gdnf family receptor alpha - 1 ( gfral )
Gdnf的神经营养作用主要是通过两类受体亚基介导，即胶质细胞源性神经营养因子受体alpha ( gfr s )亚基和受体酪氨酸激酶ret亚基。 - Pka , receptor tyrosine kinase ( trk ) and classical nuclear receptor of gc were not involved in the gc " s activation of mapks the second part studied the nuclear translocation of gc activated mapks , mainly p38 and jnk , with laser confocal microscopy . the results showed that : 1
Gc激活的mapks的激活不需要pka酪氨酸激酶受体trk及经典gc核受体的参与第二部分是研究gc激活的mapks的核转位，主要是p38和jnk ，用激光共聚焦显微镜观察到以下结果： 1